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The application of targeted IGFs in cancer therapies and related mechanisms of action are summarized in Table I

Through paracrine signaling, MenSCs increase proliferating cell nuclear antigen (PCNA) expression to promote cell proliferation, inhibit caspase-3-mediated apoptosis to reduce cell death, increase the secretion of anti-inflammatory factors such as IL-10 and keratinocyte growth factor (KGF), and simultaneously suppress proinflammatory cytokines such as IL-1, neutrophil infiltration, and myeloperoxidase (MPO) activity, thereby attenuating pulmonary inflammation and restoring lung function

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The most compelling recent evidence linking glutathione dysregulation to complex chronic illness comes from the 2024 PNAS study by Stanford University researchers

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