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A study on Grave's disease pointed out that the increased levels of I-FABP and zonulin can damage intestinal epithelial cells through direct contact, toxin release and innate immune activation, which is probably due to the imbalance of microbiota

Polymyxin B identified as an inhibitor of alternative NADH dehydrogenase and malate:quinone oxidoreductase from Gram-positive bacterium Mycobacterium smegmatis

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Third, the discovery that MOTS-c also plays roles in immune regulation, particularly in suppressing innate immune overactivation, has opened research directions in inflammation-driven aging, autoimmunity, and potentially long-term sequelae of infections