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While the genetic basis of HD is well defined, the downstream molecular mechanisms contributing to neuronal death and disease progression remain an area of active research

The challenge lies in modulating inflammation rather than eliminating it entirely

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Agitation and aggression related pathology Studies on agitation and aggression in AD have identified various neuronal and functional changes, including atrophy, low fractional anisotropy scores, increased functional connectivity, amyloid accumulation, and hypometabolism in regions such as the amygdala, anterior cingulate, hippocampus, frontal insula, and frontal cortex (Tekin et al., 2001

36, 37 Its protective role has been ascribed to the ability to inhibit the activation of the caspase cascade