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In general, three different groups of agents were described in the past: (i) mAbs, either targeting the ligand HGF or the receptor Met, (ii) tyrosine kinase inhibitors, targeting the tyrosine kinase domain of Met (and mostly also the TK domain of other RTKs), (iii) a truncated, soluble Met receptor serving as decoy for HGF (44) and the competitive HGF antagonist NK4 (18)

These complexes can deposit on hepatocyte membranes or LSECs, activating the complement system and triggering local inflammatory responses, leading to hepatocyte damage [89]

doi: 10.1080/10715769800300101

An increasing body of evidence suggests that the gut microbiota and neuroendocrine system, both of which are linked to conditions such as irritable bowel syndrome and depression, engage in bidirectional communication

population, according to an analysis of NHANES data