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Quiescent HSCs in both human and mouse and neural stem cells (NSCs) in mouse, for example, depend on the FOXO1CPT1A axis and FAO to maintain self-renewal capacity in low-nutrient or hypoxic niches 34,35

Conclusions This study has provided insights into molecular processes associated with emphysematous destruction of the lung and revealed mechanisms that contribute to the pathogenesis of COPD

We found that MitoQ was non-cytotoxic up to a concentration of 100 nM over a 24-hour treatment period ( Figure 1A )

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