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Insights into the molecular basis for substrate binding and specificity of the wild-type L-arginine/agmatine antiporter AdiC

doi: 10.1007/s10068-015-0230-9 192

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UVR disrupts microbial equilibrium through direct DNA damage and indirect release of pathogen-associated molecular patterns (PAMPs) and damage-associated molecular patterns (DAMPs), such as oxidized lipids and porphyrins, promoting pathogenic overgrowth (e.g., Staphylococcus aureus )