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Thus, DNA damage emerges as a key downstream consequence of persistent oxidative stress, linking environmental toxin exposure directly to genomic instability and mutagenesis [17,18,19]

Aside from the molecules mentioned above, several other compounds with anti-ferroptotic properties have demonstrated antifibrotic effects in various organs besides the lungs, and hold promise for potential therapeutic application in PF

Linus Pauling Institute Oregon State University Updated in April 2003 by: Jane Higdon, Ph.D

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