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Also, activation of liver GH receptor, promotes IGF-I synthesis which, in turn, is released to the circulation and can be found in its free form but mainly bound to IGFBPs (overall IGFBP-3, which binds ~90% of circulating IGF-I) [23]

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However, this becomes dysfunctional in AD, further worsening the effects of oxidative stress (Dringen and Arend, 2025)