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Proteomic analysis identified altered programmed cell death (PCD) pathways as central to resistance

Mechanistic studies revealed that AS-IV inhibits JNK signaling pathway activation by downregulating phosphorylated JNK (p-JNK) levels, thereby modulating downstream apoptosis-related molecules, such as reducing the Bax/Bcl-2 ratio and suppressing caspase-3 activation, ultimately blocking the mitochondrial apoptosis pathway

A case of a 14-year-old initially treated with clindamycin and surgical debridement, highlights the complexity of AF, including challenges in diagnosis, treatment, and the importance of early dermatological consultation

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