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The mechanism appears to involve tyrosinase activity, oxidative stress, and toxic metabolites that place melanocytes under severe pressure

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Elevated intracellular sorbitol also provides excess nicotinamide adenine dinucleotide (NADH) to the mitochondrial electron transport chain, which is a substrate for complex I (substrate for complex I) in mitochondria and closely affects mitochondrial function and cellular energy metabolism

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B(12)-dependent photoresponsive protein hydrogels for controlled stem cell/protein release