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By introducing additional A1AT through gene delivery using a recombinant adeno-associated virus, researchers were able to observe a significant reduction in insulitis and prevent the development of hyperglycemia in non-obese diabetic mice ( A notable finding in this context is the potential of A1AT to extend the survival of transplanted islet cells and regulate the immune response in a mouse model of islet cell transplantation (113, 114)

Increased MG was found in cerebrospinal fluid of patients with Alzheimers disease, and this was associated with poorer cognitive function and lower brain volume (Angeloni et al., 2014)

MZB cells are thought to develop from M B cells but also from T2 B cells 60,61

In a recent investigation undertaken by our group, adult mice with partial IGF-1 showed a decrease in the expression of genes involved in glucose metabolism (phosphoenolpyruvate carboxylase-1, glucose-6-phosphatase, pyruvate dehydrogenase kinase isoenzyme-4, and ATP-citrate lyase), resulting serious hyperglycaemia [164]

Pipeline of obesity pharmacotherapies GLP-1 receptor agonists GLP-1 RA increase satiety, reduce food intake and delay gastric emptying whilst they also stimulate insulin release and inhibit glucagon secretion in a glucose-dependent manner (Fig