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found that inhibiting -catenin expression increased alveolar remodeling and reduced the incidence of pulmonary vascular remodeling and pulmonary hypertension in newborn rats, suggesting the involvement of Wnt/-catenin pathway in hyperoxia-induced alveolar damage (61)

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The hyper-oxidation of 2-Cys Prx, in particular of Prx1, adds further chaperone function to these Prxs, but depends on certain motif elements downstream of the peroxidatic cysteine residue (GGLG and YF motifs) [23, 61]

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