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Indeed, systemic administration of clinically relevant doses of EA, a GST inhibitor [45], increased the intra-tumoural GSH levels (Figure 6c) and re-sensitised EGFRm/T790M tumours to erlotinib in a cancer cell xenograft mice model (Figure 6ac)

The method of claim 3, comprising screening the expressed antigen binding site polypeptide for its ability to specifically bind an antigen by a method comprising expression of the expressed antigen binding site polypeptide in a solid phase