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Treatment plans should be personalized and optimized based on microbial community genotyping (e.g., intervention for L

AMPK controls autophagy by suppressing mTORC1, which in turn encourages the activation of ULK1 (ULK1, ULK2, ATG13, and FIP200) (Fig

Moreover, -hydroxybutyric acid demonstrated antioxidant effects by reducing prooxidant markers of OS, including ROS and nitrite content, while elevating glutathione content, resulting in decreased lipid peroxidation

However, the inhibitory effect of N-acetylcysteine was significantly less than that observed with H5N1, suggesting the susceptibility is strain-dependent

Patients should be offered genetic counseling