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Ferroptosis in hyperosmolarity-induced HCECs was suppressed when excessive oxidative stress was mitigated

Conclusion The present study demonstrates the destructive consequences of noise stress on the brain

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Enzymatic assays To assay the activities of Cp1A and MfaA, 100-l reactions were performed at 30 C for 3 h in 50 mM sodium phosphate buffer (pH 8.0) containing 0.2 mM FeSO 4 , 2 mM KG, 2 mM ascorbate, 1 mM substrate and 10 M Cp1A or MfaA