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The limited role for NADPH partially explains why patients with glucose-6-phosphate dehydrogenase deficiency with a resultant deficiency in NADPH are not at increased risk of developing methemoglobinemia, although they are at risk of developing hemolysis following exposure to an oxidant stress

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Despite this finding, it is not clear how a single defect in peroxisomal fatty acid metabolism could elicit such a varied neurologic phenotype

Next, the neural markers are selected as feedback features and trained in the laboratory according to traditional visual or auditory NFT

BMC Cancer 14:621