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[27] demonstrated an infiltration of dT cells 3 days after the onset of ischemia in a mouse model, along with a production of IL-17 which amplify the inflammatory cascade

Conditional hepatic knockout of G3BP1 exacerbates HFD-induced MASLD and HFHC-induced MASH Considering that lipotoxicity is a major cause of MASLD initiation 27 , we conducted experiments using a 20-week high-fat diet (HFD) feeding regimen to examine the function of G3BP1 in a mouse model of MASLD

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