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Recently, evidence has emerged linking the absence of the osteocalcin gene with metabolic dysfunctions such as hyperglycemia, glucose intolerance, increased fat mass and serum triglyceride levels, and decreased production of cells, insulin secretion, and adiponectin expression

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Despite this, we and many other researchers believe that the heterogeneity of face validity in mouse models of ASD could be converted from a problem into an advantage as long as investigators exploit these differences to advance our understanding of autism