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Figure 6 Intracellular GSH Is a Regulator of BDG-Induced NO Secretion, Phagocytosis, and Cytokine Production As metabolism of myeloid cells is closely linked to their effector functions and intracellular GSH levels regulated BDG-induced metabolic changes, we wanted to investigate if GSH could also regulate monocytes effector functions in response to BDG

Mechanisms of action of incretin receptor based dual- and tri-agonists in pancreatic islets

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