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This regulatory mechanism in H1299 cells was further validated, as western blot analysis confirmed that the methionine-SAM axis controls ACSL4 protein abundance possibly through methylation modifications (Fig

Younger patients were more likely to have primary AH, while older patients were more likely to have secondary AH

Therapeutic effects in patient-derived osteosarcoma xenograft mouse model For a better understanding of the clinical potential, its therapeutic properties were assessed in a patient-derived osteosarcoma xenograft mouse model (PDX OS ) in immunodeficient nude mice (protocol for model establishment: Fig

Many disease-causing mutations cluster near the structural NADP binding site, destabilizing the enzyme without directly abolishing its active site

Weight of evidence: Overall evidence is Low : primarily preclinical with heterogeneous designs and no consistent human biomarkers