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The loss of Mfn2 has been shown to cause an increase in unopposed Drp1 activity and subsequent increased mitochondrial fragmentation and ROS production

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Looking ahead, stimuli-responsive hydrogels capable of on-demand pulsatile PRLM release, particularly when integrated with personalized devices such as microneedles or cardiac patches, represent an innovative strategy for advancing tissue repair and cardiovascular therapy

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However, approximately one-third of patients needing treatment may not receive treatment [3], and 3560% of treated patients may not achieve clinical remission [4]