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[DOI] [PubMed] [Google Scholar] 106.Conrad M., Kagan V.E., Bayir H., Pagnussat G.C., Head B., Traber M.G., Stockwell B.R

Accordingly, missense DNA mutations that impair G6PD activity or stability result in increased oxidative stress and a spectrum of disease phenotypes featuring, most commonly, hemolytic anemia, and collectively called G6PD deficiency 1

High levels of sugar in the blood (hyperglycemia) and insulin deficiency or resistance can lead to increased oxidative stress

Ferroptotic injury subsequently compromises BRB integrity, exacerbates iron accumulation and lipofuscin deposition, and culminates in retinal nerve fiber layer thinning and visual dysfunction

DNA takes tens of thousands of oxidative moments every day, and antioxidants are what counteracts the damage associated with free radicals