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Treatment with inhibitor ferrostatin-1 (Fer-1) effectively suppresses high hydrostatic pressure-induced VSMC inflammation, as evidenced by reduced CXCL2 expression, and prevented endothelial dysfunction-associated phenotype switching marked by decreased AKR1C2 expression

It contributes to the reduction of fatigue and exhaustion

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N-acetylcysteine inhibits muscle fatigue in humans

The hydrogel, when covering wound surfaces, is susceptible to damage from skin movement, which could impede wound healing and increase infection risk