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It also predicted the phenotype of experiment of nature in humans with gene defects in the axis yet to be discovered

found that in the early stage of exposure to acute hypobaric hypoxia, the stress of plasma H2S production was increased and pulmonary arterial pressure was elevated, suggesting that exogenous supplementation of H2S could delay acute hypobaric hypoxia-induced pulmonary arterial pressure elevation (Yang et al., 2011)