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Thus, when 2 binds to the GD of AAGAB, the resulting AAGAB:2 dimer represents a dead-end configuration that cannot recruit , providing a molecular explanation for why this binding is not observed in cells (Fig

doi: 10.1080/19490976.2023.2185034 526 GargVVelumaniDLinYCHayeA

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Common biological features of chronic injury Microvascular limitation: the tissue is under-perfused, which slows nutrient delivery and waste removal Inflammatory persistence: cytokine signaling stays elevated and disrupts remodeling Disorganized collagen: collagen exists, but it is poorly aligned, weak, or prone to reinjury Mechanical mismatch: loads exceed tissue capacity due to weakness, compensation, or instability Neurogenic sensitivity: irritated tissues and nerves amplify pain signals even when imaging is normal In other words, chronic injuries do not usually need one intervention

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