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We report here a multi-omics analysis in patients with ME/CFS and healthy controls (HC), before and 24 h after exercise, that suggests models for understanding the pathogenesis of chronic inflammation, fatigue, and PEM in ME/CFS

To confirm these results, we knockeddown unc1 expression in a tissuespecific manner using RNAi by expressing doublestranded RNAs against the gene across the entire nervous system ( rab3 promoter) using transgenesis (Appendix Fig S8)

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Zhang CH, Wang Y, Sun QQ, Xia LL, Hu JJ, Cheng K, Wang X, Fu XX, Gu H (2018) Copper nanoparticles show obvious in vitro and in vivo reproductive toxicity via ERK mediated signaling pathway in female mice

Mechanistically, oxidative stress triggers endoplasmic reticulum stress through the activation of the PERK-eIF2-ATF4 and IRE1-XBP1 signaling pathways, as well as through the upregulation of CHOP (C/EBP homologous protein), which promotes apoptotic signaling (75) as shown in Figure 4