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Administering sodium hydrosulfide (NaSH), at 2.8 mg/kg, once a day for 3 months ameliorated memory deficits, reduced APP and BACE1, upregulated the master regulator of antioxidant response genes, nuclear factor erythroid-2-related factor 2 (Nrf2), heme oxygenase-1(HO-1) and glutathione S-transferase (GST) in the APP/PS1 model of AD (Liu et al., 2 S donor, AP-39, maintained cellular bioenergetics and preserved mitochondrial function in the APP/PS1 mouse model of AD (Zhao et al., 2016)

This approach was recently used to identify the molecular determinants of the Warburg effect 11 and to explore novel therapeutic strategies for cancer such as metabolic synthetic lethality 12

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