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Huntingtons disease

By secreting SASP factors such as CCL2, they recruit and activate immune cells and fibroblasts within the stroma [126, 127]

Small-molecule inhibitors of cGAS enzymatic activity (e.g., RU.521, G140, PF-06928215) or STING signaling (e.g., H-151, C-176) have shown promise in preclinical neuroinflammation and neurodegeneration models by suppressing excessive type I interferon and cytokine production [12, 196]