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The net effect was convergence toward a youthful phenotype regardless of starting point

After adjustment for multiple clinical variables, neither variability nor CoV of LS were predictors of transplant-free survival (TFS) or hepatic decompensation (HD)

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However, rT3 is active in the cytoplasm of neurons and glial cells in the brain where it and T4 bind a cytoplasmic truncated thyroid receptor to trigger actin-cytoskeletal polymerization, which is important for brain development and migration of neurites [1]