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In AD animal models, M1-polarized microglia frequently co-exist with iron accumulation, indicating a synergistic interaction between inflammatory activation and disrupted iron metabolism in driving neurotoxicity (Mendoza and Scharff, 2017)

investigated the warm and cold types of AIHA (wAIHA and cAIHA, respectively) and demonstrated that eryptosis is predominantly driven by cold IgM and/or IgA antibodies rather than warm IgG autoantibodies, as shown by increased PS exposure in response to autoantibodies [329]