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The study found that this pathway begins with the activation of c-Jun-N-terminal kinase 1 (JNK1) and increased receptor-interacting protein 1 levels (RIP-1)

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Similarly, a roGFP2-iL-Grx1 fusion redox probe targeted to the ER of Arabidopsis thaliana cells lacking Ero1/2, the functional homolog of DsbB in eukaryotes was more reduced, and re-oxidation after a reductive pulse was inhibited by the lack of Ero1/2 (Ugalde et al., 2022)

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