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NADPH oxidase (NOX), lipoxygenase, cyclooxygenase (COX), xanthine oxidase (XO), uncoupled nitric oxide synthases (NOS), cytochrome P450, and mitochondrial respiration are the most common enzymatic causes of ROS in CVD [11]

Loss of PFK resulted in a decrease of FA abundance and a concomitant reduction in cell size, that is, in a phenotype opposite to that of aldolase depletion, whereas GAPDH knockdown did not result in any adhesion phenotype (Fig