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The present review highlights disrupted mitochondrial homeostasis, including impaired OXPHOS, calcium imbalance, redox stress and defective mitophagy, as a central mechanism linking E2 loss to neuropsychiatric symptoms

Control cells were: PHM141 (human myometrial myocytes), MS1 (mouse pancreatic islet endothelium), Ishikawa (human endometrial adenocarcinoma), and HeLa cells (cytokeratin only)