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Given NR3C1s central role in HPA axis regulation and its known involvement in depression, such epigenetic dysregulation may represent a shared vulnerability mechanism across addiction and mood disorders

It is worth stressing that neither modification was intended to make the molecule better in any therapeutic sense

(2011) 127:78794.e1-9

Therefore, decreased NCOA4 expression and impaired binding of NCOA4 to FTH1 diminish the FTH1 degradation by downregulating ferritinophagy, and decreased FPN1 expression inhibits iron output and release to other tissues, both of which effectively increase liver iron overload and, in turn, trigger oxidative stress and ferroptosis (82)

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