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& Weber, T

Do not fight it

Reduced myostatin signaling in preclinical MOTS-c work

Arrhythmias are most common in patients with diseased or deformed hearts, often due to congenital heart defects, and they are unlikely to develop in patients with healthy hearts unless they are triggered by an outside factor, such as electrical shock or illicit drug use

We suggest glutathione is one such endogenous source, as impairing glutamate liberation from the cycle further diminishes synaptic activity by MeAIB, while increasing glutamate availability can rescue the impairment by MeAIB