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Documentation of iron deficiency without hematologic evidence of such (hypochromasia in blood smears, low MCV, low MCHC) is difficult because serum iron concentrations are not the best surrogate of iron stores and ferritin, the iron storage protein, can be increased by inflammation

Zinc acetate, trientine, D-penicillamine, and tetrathiomolybdate are used for the treatment

Mitotic cell lysates were immunoblotted to assess if deletion of CEP192 (exon 11) also had a measurable impact on whole cellular, and not only spindle-bound, levels of total and autophosphorylated Aurora-A

or fatigability in adulthood (34, 35)