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Small SALT TOLERANCE ENHANCER1 (STE1) protein promotes ABA-dependent salt stress-responsive pathways by interacting with the ABA receptor PYLs and the positive ABA signaling regulator SnRK2s and by improving Na + and K + homeostasis and ROS scavenging (Meng et al., 2020)

5) and can be started regardless of the type of enzyme deficiency underlying the urea cycle [38, 39], the goal of treatment being to reduce ammonia production and accelerate elimination via alternative pathways (Fig

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BPC-157 targets vascular and growth factor systems