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Advances in the delivery of RNA therapeutics: from concept to clinical reality

Another mechanism by which pepsin can perpetuate its harmful effects is by reducing carbonic anhydrase-III levels, which play a fundamental role in local protection against the deleterious effects of stomach acid content (Reference Gill, Johnston and Buda34)

Terminal endpoints included plasma biomarkers alanine/aspartate aminotransferase (ALT/AST), metallopeptidase inhibitor 1 (TIMP-1) and amino terminal peptide of type III procollagen (PIIINP), liver biochemistry, NAFLD Activity Score (NAS), fibrosis stage and quantitative liver histology

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Expanding adipose depots develop localized hypoxia that activates hypoxia-inducible factor 1- (HIF-1)-dependent signaling cascades, establishing self-perpetuating cycles of matrix accumulation