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This effect was associated with modulation of mTOR-dependent signaling and restoration of autophagic flux, thereby promoting adaptive cellular repair rather than maladaptive cell death (Guo et al

High doses of N-acetylcysteine alone or in combination with inhaled corticosteroids and oxidative stress in patients with COPD [abstract]

In agreement, SLC7A11 knockdown in ECs increased the expression of FN1 and SNAI1 but decreased that of CDH5 and NR2F2 (Figure 4H), indicative of augmented endothelial-to-mesenchymal transition (EndoMT)

During this stage, the liver renders reactive intermediate metabolites less harmful by chemically binding them to water-soluble compounds, a process known as conjugation

10.1242/jcs.206979 105 QuimbyJ