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Whether this disparity is due to the sensitivity of assessments or a weaker influence of the cerebellum on cognitive processes is still uncertain

But I do think its important not to get stuck in overemphasizing them because you could have heterogeneric or highly variable manifestations of something, but you could have a very homogeneous or very sort of universal underlying feature of whats driving all of it

In the chronic window ( week 3) focus turns to maintenance/remodelling: preventing relapse, limiting fibrosis and endothelial dysfunction and leveraging lifestyle hormesis alongside gentle, compartment-targeted pharmacology

Peptidomimetics via modifications of amino acids and peptide bonds

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