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Therefore, it can be speculated that Gln deprivation may inhibit IL-17 production by suppressing the IL-23-STAT3 pathway ( Earlier studies demonstrated that serum -KG and Glu were abnormally elevated in patients with psoriasis (7375)

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These changes are directly responsible for the phenomenon of fetal immunotolerance, inhibiting the development and activation of a subpopulation of T lymphocytes that can stimulate inflammatory reactions within the uterine endometrium, while maintaining the endometrial and trophoblast defense against pathogens (165)

these, together with more recent work 18,22 , reveal substantial differences in the rates of reaction of HCHO with nucleobases, and in the stabilities of the HCHO-nucleobase adducts (including when produced by enzymatic oxidation of N -methyl groups)

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