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In HCC cells, ATB0,+ and ASCT2 collectively account for the majority of Gln uptake

However, the processes that are responsible for GSSG accumulation when CAT2 function is lost remain unclear

These chronic attributes are generally associated with selective degeneration of dopaminergic neurons, corollary de-pigmentation of substantia nigra pars compacta (SNpc) and concomitant accumulation of -synuclein in insoluble inclusions called Lewy bodies 2,3