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2020;40:226-230 Smedberg M

Elevated levels of TNF- in the AH of individuals with glaucoma, compared to controls, facilitate the apoptosis of RGCs via interaction with TNF-receptor-1 (TNF-R1)

This process is directed by peptide sequences around the cysteine residue, exogenous NO sources, or by compartmentalization of NO synthesis

Nevertheless, these gains are less marked than those achieved with estrogen or bisphosphonates, being associated with a relatively high incidence of adverse effects, including the development of peripheral edema and carpal tunnel syndrome (Holloway et al., 1997)