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Consequently, ferroptosis, cuproptosis, and disulfidptosis may involve interrelated crosstalk mechanisms involving glycolysis and oxidative phosphorylation in the context of circulatory disturbance

CJC-1295 may stimulate fat loss: Studies have shown that prolactin promotes visceral fat accrual in various animal models [12] and that CJC-1295 can normalize prolactin levels as shown in GHRH knockout mice [13], which can lead to increased fat metabolization

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Beyond gastric cancer, nanotechnology-enabled therapeutic strategies are being actively explored across gastrointestinal malignancies as part of a broader translational movement toward programmable drug delivery and microenvironment-aware therapy