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The effect of reactive astrogliosis after acute injury depends on the presence of the ischemic penumbra and differs between the immature and adult CNS

Gut microbiota drives macrophage-dependent self-renewal of intestinal stem cells via niche enteric serotonergic neurons

When external stimuli excite the vagus nerve, the nerve terminals produce the anti-inflammatory parasympathetic neurotransmitter acetylcholine (ACh), which activates the 7 nicotinic acetylcholine receptors (7nAChR) on monocytes and macrophages

Inhibiting TGF- could reduce fibrosis but may interfere with its protective roles in resolving inflammation and promoting tissue repair, leading to unintended consequences.93 94 Therefore, therapeutic approaches targeting TGF- must be carefully considered and finely tuned to balance its profibrotic and anti-inflammatory effects

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