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Although the etiologies of most patients are still unknown, ASD exhibits clinical variability and can co-occur in up to 10% of cases with well-characterized neurological and genetic abnormalities, including co-occurring syndromes (Rapin, 2002)

This is due to the fact that GHK is able to inactivate the damaging byproducts released from free radical reactions

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Depending on the availability of cellular GSH and the activation of glutathione S-transferase subclasses, detoxification of AFB1 is facilitated [24]