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However, this report highlights that patients with compatible symptoms and exposure histories may not fit neatly into laboratory definitions

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Although research into this syndrome is increasing, the aetiology is not clear, although the symptoms are thought to be due to nociceptive or neurogenic mechanisms

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These recurrent waves of increased concentrations of K + and astrocytic Ca 2+ cause cytotoxic edema (Hartings et al., 2017) and may potentially result in terminal depolarizations and falls in perfusion, causing secondary expansion of the infarct core at the expense of the penumbral region (Strong et al., 2007