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Conclusion: While a significant increase in Ybx1 expression in differentiating preadipocytes may support healthy adipogenesis, our findings reveal a detrimental mechanism in which the maladaptive activation of Ybx1 within hepatocytes directly stimulates the C/EBP:PPAR pathway, leading to an increase in adipogenic gene expression that becomes lipotoxic within the liver

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The majority of patients had non-alcoholic steatohepatitis (NASH, 36%) or combination of alcohol-related liver disease and NASH (27%)

We used linear regression analyses to examine the associations of patients self-reported health status (terminally ill vs

doi:10.1002/ncp.10234 [24] Ringseis R, Keller J, Eder K