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NLRP3 knock-out reduced these effects, suggesting that an IL-1-dependent NLRP3/caspase-1 neuroimmune pathway is a key driver of fatigue and providing a rationale for targeting this axis therapeutically [77]

Sample size in the hundreds, follow-up of several months, prespecified primary endpoint, independent funding or at least transparent funding disclosure, replication of key findings

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Molecular cloning, characterization, and expression in Escherichia coli of full-length cDNAs of three human glutathione S-transferase Pi gene variants