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Indian J Ophthalmol 68(7):1448-1449, 2020

The absence of smad4 was also found to enhance mitochondrial ATP synthase inhibitory factor (ATPIF1) activity, ATP synthase inhibition, and reduce mitochondrial membrane potential, ultimately promoting glycolysis by decreasing glucose metabolic flux and increasing glucose accumulation in the podocyte [62]

however, it can also reduce levels of pro-inflammatory tumor necrosis factor-alpha (TNF-) and interleukins (IL-6 and IL-1), inhibit the formation of microbial biofilms and destroy biofilms, and break down disulfide bonds between mucin molecules

2012), which include DNA metabolism proteins (Gari et al

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